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Parte de monografia/livro
What is the transcriptome and how it is evaluated (2022)
Assis, Amanda F.; Oliveira, Ernna Hérida Domingues de; Donate, Paula Barbim; Giuliatti, Silvana ; Nguyen, Catherine; Passos Junior, Geraldo Aleixo da Silva
Fonte: Transcriptomics in health and disease. Unidades: FMRP, FORP
Assuntos: RNA MENSAGEIRO, SEQUENCIAMENTO GENÉTICO, ANÁLISE EM MICROSSÉRIES, GENOMAS, EXPRESSÃO GÊNICA
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ABNT
ASSIS, Amanda F. et al. What is the transcriptome and how it is evaluated. Transcriptomics in health and disease. Tradução . Cham: Springer, 2022. . Disponível em: https://doi.org/10.1007/978-3-030-87821-4_1. Acesso em: 17 maio 2024.
APA
Assis, A. F., Oliveira, E. H. D. de, Donate, P. B., Giuliatti, S., Nguyen, C., & Passos Junior, G. A. da S. (2022). What is the transcriptome and how it is evaluated. In Transcriptomics in health and disease. Cham: Springer. doi:10.1007/978-3-030-87821-4_1
NLM
Assis AF, Oliveira EHD de, Donate PB, Giuliatti S, Nguyen C, Passos Junior GA da S. What is the transcriptome and how it is evaluated [Internet]. In: Transcriptomics in health and disease. Cham: Springer; 2022. [citado 2024 maio 17 ] Available from: https://doi.org/10.1007/978-3-030-87821-4_1
Vancouver
Assis AF, Oliveira EHD de, Donate PB, Giuliatti S, Nguyen C, Passos Junior GA da S. What is the transcriptome and how it is evaluated [Internet]. In: Transcriptomics in health and disease. Cham: Springer; 2022. [citado 2024 maio 17 ] Available from: https://doi.org/10.1007/978-3-030-87821-4_1
Artigo de periodico
Curcumin-loaded carrageenan nanoparticles: fabrication, characterization, and assessment of the effects on osteoblasts mineralization (2022)
Nogueira, Lucas Fabricio Bahia ; Cruz, Marcos Antônio Eufrasio ; Tovani, Camila Bussola; Lopes, Helena Bacha ; Beloti, Márcio Mateus ; Ciancaglini, Pietro ; Bottini, Massimo; Ramos, Ana Paula
Fonte: Colloids and Surfaces B: Biointerfaces. Unidades: FORP, FFCLRP
Assuntos: NANOPARTÍCULAS, MEDICAMENTO, OSTEOCLASTO
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NOGUEIRA, Lucas Fabricio Bahia et al. Curcumin-loaded carrageenan nanoparticles: fabrication, characterization, and assessment of the effects on osteoblasts mineralization. Colloids and Surfaces B: Biointerfaces, v. 217, p. 1-9, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.colsurfb.2022.112622. Acesso em: 17 maio 2024.
APA
Nogueira, L. F. B., Cruz, M. A. E., Tovani, C. B., Lopes, H. B., Beloti, M. M., Ciancaglini, P., et al. (2022). Curcumin-loaded carrageenan nanoparticles: fabrication, characterization, and assessment of the effects on osteoblasts mineralization. Colloids and Surfaces B: Biointerfaces, 217, 1-9. doi:10.1016/j.colsurfb.2022.112622
NLM
Nogueira LFB, Cruz MAE, Tovani CB, Lopes HB, Beloti MM, Ciancaglini P, Bottini M, Ramos AP. Curcumin-loaded carrageenan nanoparticles: fabrication, characterization, and assessment of the effects on osteoblasts mineralization [Internet]. Colloids and Surfaces B: Biointerfaces. 2022 ; 217 1-9.[citado 2024 maio 17 ] Available from: https://doi.org/10.1016/j.colsurfb.2022.112622
Vancouver
Nogueira LFB, Cruz MAE, Tovani CB, Lopes HB, Beloti MM, Ciancaglini P, Bottini M, Ramos AP. Curcumin-loaded carrageenan nanoparticles: fabrication, characterization, and assessment of the effects on osteoblasts mineralization [Internet]. Colloids and Surfaces B: Biointerfaces. 2022 ; 217 1-9.[citado 2024 maio 17 ] Available from: https://doi.org/10.1016/j.colsurfb.2022.112622
Artigo de periodico
The chemically-modified tetracycline COL-3 and its parent compound doxycycline prevent microglial inflammatory responses by reducing glucose-mediated oxidative stress (2021)
Ferreira Junior, Nilson Carlos; Pereira, Maurício dos Santos ; Francis, Nour; Ramirez, Paola; Martorell, Paula; González-Lizarraga, Florencia; Figadère, Bruno; Chehin, Rosana; Del Bel, Elaine Aparecida ; Raisman-Vozari, Rita; Michel, Patrick Pierre
Fonte: Cells. Unidade: FORP
Assuntos: ESTRESSE OXIDATIVO, GLICOSE, METABOLISMO, ANTIBIÓTICOS, AGENTES ANTIMICROBIANOS, FARMACOLOGIA EXPERIMENTAL
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FERREIRA JUNIOR, Nilson Carlos et al. The chemically-modified tetracycline COL-3 and its parent compound doxycycline prevent microglial inflammatory responses by reducing glucose-mediated oxidative stress. Cells, v. 10, n. 8, p. 1-16, 2021Tradução . . Disponível em: https://doi.org/10.3390/cells10082163. Acesso em: 17 maio 2024.
APA
Ferreira Junior, N. C., Pereira, M. dos S., Francis, N., Ramirez, P., Martorell, P., González-Lizarraga, F., et al. (2021). The chemically-modified tetracycline COL-3 and its parent compound doxycycline prevent microglial inflammatory responses by reducing glucose-mediated oxidative stress. Cells, 10( 8), 1-16. doi:10.3390/cells10082163
NLM
Ferreira Junior NC, Pereira M dos S, Francis N, Ramirez P, Martorell P, González-Lizarraga F, Figadère B, Chehin R, Del Bel EA, Raisman-Vozari R, Michel PP. The chemically-modified tetracycline COL-3 and its parent compound doxycycline prevent microglial inflammatory responses by reducing glucose-mediated oxidative stress [Internet]. Cells. 2021 ; 10( 8): 1-16.[citado 2024 maio 17 ] Available from: https://doi.org/10.3390/cells10082163
Vancouver
Ferreira Junior NC, Pereira M dos S, Francis N, Ramirez P, Martorell P, González-Lizarraga F, Figadère B, Chehin R, Del Bel EA, Raisman-Vozari R, Michel PP. The chemically-modified tetracycline COL-3 and its parent compound doxycycline prevent microglial inflammatory responses by reducing glucose-mediated oxidative stress [Internet]. Cells. 2021 ; 10( 8): 1-16.[citado 2024 maio 17 ] Available from: https://doi.org/10.3390/cells10082163
Artigo de periodico
Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of parkinson’s disease (2021)
Pereira, Maurício dos Santos ; Abreu, Gabriel Henrique Dias; Rocca, Jeremy; Hamadat, Sabah; Raisman-Vozari, Rita; Michel, Patrick Pierre; Del Bel, Elaine Aparecida
Fonte: Frontiers in Pharmacology. Unidade: FORP
Assuntos: ASTROCITOMA, CANABINOIDES, GLUTAMATOS
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PEREIRA, Maurício dos Santos et al. Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of parkinson’s disease. Frontiers in Pharmacology, v. 11, p. 1-16, 2021Tradução . . Disponível em: https://doi.org/10.3389/fphar.2020.617085. Acesso em: 17 maio 2024.
APA
Pereira, M. dos S., Abreu, G. H. D., Rocca, J., Hamadat, S., Raisman-Vozari, R., Michel, P. P., & Del Bel, E. A. (2021). Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of parkinson’s disease. Frontiers in Pharmacology, 11, 1-16. doi:10.3389/fphar.2020.617085
NLM
Pereira M dos S, Abreu GHD, Rocca J, Hamadat S, Raisman-Vozari R, Michel PP, Del Bel EA. Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of parkinson’s disease [Internet]. Frontiers in Pharmacology. 2021 ; 11 1-16.[citado 2024 maio 17 ] Available from: https://doi.org/10.3389/fphar.2020.617085
Vancouver
Pereira M dos S, Abreu GHD, Rocca J, Hamadat S, Raisman-Vozari R, Michel PP, Del Bel EA. Contributive role of TNF-α to L-DOPA-induced dyskinesia in a unilateral 6-OHDA lesion model of parkinson’s disease [Internet]. Frontiers in Pharmacology. 2021 ; 11 1-16.[citado 2024 maio 17 ] Available from: https://doi.org/10.3389/fphar.2020.617085
Artigo de periodico
Cannabidiol prevents LPS-induced microglial inflammation by inhibiting ROS/NF-κB-dependent signaling and glucose consumption (2020)
Pereira, Maurício dos Santos; Guimarães, Francisco Silveira ; Del Bel, Elaine Aparecida ; Raisman-Vozari, Rita; Michel, Patrick P.
Fonte: Glia. Unidades: FMRP, FORP
Assuntos: CANABINOIDES, GLICOSE, ESTRESSE OXIDATIVO
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PEREIRA, Maurício dos Santos et al. Cannabidiol prevents LPS-induced microglial inflammation by inhibiting ROS/NF-κB-dependent signaling and glucose consumption. Glia, v. 68, n. 3, p. 561-573, 2020Tradução . . Disponível em: https://doi.org/10.1002/glia.23738. Acesso em: 17 maio 2024.
APA
Pereira, M. dos S., Guimarães, F. S., Del Bel, E. A., Raisman-Vozari, R., & Michel, P. P. (2020). Cannabidiol prevents LPS-induced microglial inflammation by inhibiting ROS/NF-κB-dependent signaling and glucose consumption. Glia, 68( 3), 561-573. doi:10.1002/glia.23738
NLM
Pereira M dos S, Guimarães FS, Del Bel EA, Raisman-Vozari R, Michel PP. Cannabidiol prevents LPS-induced microglial inflammation by inhibiting ROS/NF-κB-dependent signaling and glucose consumption [Internet]. Glia. 2020 ; 68( 3): 561-573.[citado 2024 maio 17 ] Available from: https://doi.org/10.1002/glia.23738
Vancouver
Pereira M dos S, Guimarães FS, Del Bel EA, Raisman-Vozari R, Michel PP. Cannabidiol prevents LPS-induced microglial inflammation by inhibiting ROS/NF-κB-dependent signaling and glucose consumption [Internet]. Glia. 2020 ; 68( 3): 561-573.[citado 2024 maio 17 ] Available from: https://doi.org/10.1002/glia.23738
Artigo de periodico
Rifampicin and its derivative Rifampicin quinone reduce microglial inflammatory responses and neurodegeneration induced in vitro by α-Synuclein fibrillary aggregates (2019)
Acuña, Leonardo; Hamadat, Sabah; Corbalán, Natalia S.; González-Lizárraga, Florencia; Pereira, Maurício dos Santos ; Rocca, Jérémy; Díaz, Julia Sepúlveda; Del-Bel, Elaine Aparecida; Papy-García, Dulce; Chehín, Rosana N.; Michel, Patrick P.; Raisman-Vozari, Rita
Fonte: Cells. Unidade: FORP
Assuntos: DOENÇA DE PARKINSON, INFLAMAÇÃO, CITOCINAS, NEURÔNIOS, ANTIBIÓTICOS
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ACUÑA, Leonardo et al. Rifampicin and its derivative Rifampicin quinone reduce microglial inflammatory responses and neurodegeneration induced in vitro by α-Synuclein fibrillary aggregates. Cells, v. 8, n. 8, p. [17] , 2019Tradução . . Disponível em: https://doi.org/10.3390/cells8080776. Acesso em: 17 maio 2024.
APA
Acuña, L., Hamadat, S., Corbalán, N. S., González-Lizárraga, F., Pereira, M. dos S., Rocca, J., et al. (2019). Rifampicin and its derivative Rifampicin quinone reduce microglial inflammatory responses and neurodegeneration induced in vitro by α-Synuclein fibrillary aggregates. Cells, 8( 8), [17] . doi:10.3390/cells8080776
NLM
Acuña L, Hamadat S, Corbalán NS, González-Lizárraga F, Pereira M dos S, Rocca J, Díaz JS, Del-Bel EA, Papy-García D, Chehín RN, Michel PP, Raisman-Vozari R. Rifampicin and its derivative Rifampicin quinone reduce microglial inflammatory responses and neurodegeneration induced in vitro by α-Synuclein fibrillary aggregates [Internet]. Cells. 2019 ; 8( 8): [17] .[citado 2024 maio 17 ] Available from: https://doi.org/10.3390/cells8080776
Vancouver
Acuña L, Hamadat S, Corbalán NS, González-Lizárraga F, Pereira M dos S, Rocca J, Díaz JS, Del-Bel EA, Papy-García D, Chehín RN, Michel PP, Raisman-Vozari R. Rifampicin and its derivative Rifampicin quinone reduce microglial inflammatory responses and neurodegeneration induced in vitro by α-Synuclein fibrillary aggregates [Internet]. Cells. 2019 ; 8( 8): [17] .[citado 2024 maio 17 ] Available from: https://doi.org/10.3390/cells8080776
Artigo de periodico
The Autoimmune Regulator (Aire) transactivatesHLA-Ggeneexpression in thymic epithelial cells (2019)
Melo-Lima, Breno Luiz ; Poras, Isabelle; Passos, Geraldo Aleixo Silva ; Carosella, Edgardo D.; Donadi, Eduardo Antônio ; Moreau, Philippe
Fonte: Immunology. Unidades: FORP, FMRP
Assuntos: TIMO (SISTEMAS SANGUÍNEO E IMUNE), AUTOIMUNIDADE, ANTÍGENOS
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MELO-LIMA, Breno Luiz et al. The Autoimmune Regulator (Aire) transactivatesHLA-Ggeneexpression in thymic epithelial cells. Immunology, v. 158, p. 121-135, 2019Tradução . . Disponível em: https://doi.org/10.1111/imm.13099. Acesso em: 17 maio 2024.
APA
Melo-Lima, B. L., Poras, I., Passos, G. A. S., Carosella, E. D., Donadi, E. A., & Moreau, P. (2019). The Autoimmune Regulator (Aire) transactivatesHLA-Ggeneexpression in thymic epithelial cells. Immunology, 158, 121-135. doi:10.1111/imm.13099
NLM
Melo-Lima BL, Poras I, Passos GAS, Carosella ED, Donadi EA, Moreau P. The Autoimmune Regulator (Aire) transactivatesHLA-Ggeneexpression in thymic epithelial cells [Internet]. Immunology. 2019 ; 158 121-135.[citado 2024 maio 17 ] Available from: https://doi.org/10.1111/imm.13099
Vancouver
Melo-Lima BL, Poras I, Passos GAS, Carosella ED, Donadi EA, Moreau P. The Autoimmune Regulator (Aire) transactivatesHLA-Ggeneexpression in thymic epithelial cells [Internet]. Immunology. 2019 ; 158 121-135.[citado 2024 maio 17 ] Available from: https://doi.org/10.1111/imm.13099
Artigo de periodico
Repurposing an established drug: an emerging role for methylene blue in L‐DOPA‐induced dyskinesia (2019)
Santos, Keila Dayane Bariotto dos; Padovan‐Neto, Fernando Eduardo; Bortolanza, Mariza; Pereira, Maurício dos Santos; Raisman-Vozari, Rita; Tumas, Vitor; Del Bel, Elaine Aparecida
Fonte: European Journal of Neuroscience. Unidades: FMRP, FORP
Assuntos: DOENÇA DE PARKINSON, AZUL DE METILENO, ÓXIDO NÍTRICO
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SANTOS, Keila Dayane Bariotto dos et al. Repurposing an established drug: an emerging role for methylene blue in L‐DOPA‐induced dyskinesia. European Journal of Neuroscience, v. 49, n. 6, p. 869–882, 2019Tradução . . Disponível em: https://doi.org/10.1111/ejn.14079. Acesso em: 17 maio 2024.
APA
Santos, K. D. B. dos, Padovan‐Neto, F. E., Bortolanza, M., Pereira, M. dos S., Raisman-Vozari, R., Tumas, V., & Del Bel, E. A. (2019). Repurposing an established drug: an emerging role for methylene blue in L‐DOPA‐induced dyskinesia. European Journal of Neuroscience, 49( 6), 869–882. doi:10.1111/ejn.14079
NLM
Santos KDB dos, Padovan‐Neto FE, Bortolanza M, Pereira M dos S, Raisman-Vozari R, Tumas V, Del Bel EA. Repurposing an established drug: an emerging role for methylene blue in L‐DOPA‐induced dyskinesia [Internet]. European Journal of Neuroscience. 2019 ; 49( 6): 869–882.[citado 2024 maio 17 ] Available from: https://doi.org/10.1111/ejn.14079
Vancouver
Santos KDB dos, Padovan‐Neto FE, Bortolanza M, Pereira M dos S, Raisman-Vozari R, Tumas V, Del Bel EA. Repurposing an established drug: an emerging role for methylene blue in L‐DOPA‐induced dyskinesia [Internet]. European Journal of Neuroscience. 2019 ; 49( 6): 869–882.[citado 2024 maio 17 ] Available from: https://doi.org/10.1111/ejn.14079
Artigo de periodico
Relationship between human immunodeficiency virus (HIV-1) infection and chronic periodontitis (2018)
Pólvora, Tábata Larissa Santos; Nobre, Átila Vinícius Vitor; Tirapelli, Camila ; Taba Júnior, Mário ; Macedo, Leandro Dorigan de; Santana, Rodrigo de Carvalho ; Pozzetto, Bruno ; Lourenço, Alan Grupioni ; Motta, Ana Carolina Fragoso
Fonte: Expert Review of Clinical Immunology. Unidades: FORP, FMRP, FO
Assuntos: INFECÇÕES POR HIV, DOENÇAS PERIODONTAIS, SALIVA, ANTIRRETROVIRAIS, BIOMARCADORES, PERIODONTITE CRÔNICA
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PÓLVORA, Tábata Larissa Santos et al. Relationship between human immunodeficiency virus (HIV-1) infection and chronic periodontitis. Expert Review of Clinical Immunology, v. 14, n. 4, p. 315-327, 2018Tradução . . Disponível em: https://doi.org/10.1080/1744666x.2018.1459571. Acesso em: 17 maio 2024.
APA
Pólvora, T. L. S., Nobre, Á. V. V., Tirapelli, C., Taba Júnior, M., Macedo, L. D. de, Santana, R. de C., et al. (2018). Relationship between human immunodeficiency virus (HIV-1) infection and chronic periodontitis. Expert Review of Clinical Immunology, 14( 4), 315-327. doi:10.1080/1744666x.2018.1459571
NLM
Pólvora TLS, Nobre ÁVV, Tirapelli C, Taba Júnior M, Macedo LD de, Santana R de C, Pozzetto B, Lourenço AG, Motta ACF. Relationship between human immunodeficiency virus (HIV-1) infection and chronic periodontitis [Internet]. Expert Review of Clinical Immunology. 2018 ; 14( 4): 315-327.[citado 2024 maio 17 ] Available from: https://doi.org/10.1080/1744666x.2018.1459571
Vancouver
Pólvora TLS, Nobre ÁVV, Tirapelli C, Taba Júnior M, Macedo LD de, Santana R de C, Pozzetto B, Lourenço AG, Motta ACF. Relationship between human immunodeficiency virus (HIV-1) infection and chronic periodontitis [Internet]. Expert Review of Clinical Immunology. 2018 ; 14( 4): 315-327.[citado 2024 maio 17 ] Available from: https://doi.org/10.1080/1744666x.2018.1459571
Artigo de periodico
Cannabidiol prevents haloperidol-induced vacuos chewing movements and inflammatory changes in mice via PPARγ receptors (2018)
Sonego, Andreza B.; Prado, Douglas S.; Vale, Gabriel T.; Sepulveda-Diaz, Julia E.; Cunha, Thiago Mattar; Tirapelli, Carlos Renato; Del Bel, Elaine A.; Raisman-Vozari, Rita; Guimarães, Francisco Silveira
Fonte: Brain, Behavior, and Immunity. Unidades: FMRP, EERP, FORP
Assuntos: CANABINOIDES, INFLAMAÇÃO, ESTRESSE OXIDATIVO
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SONEGO, Andreza B. et al. Cannabidiol prevents haloperidol-induced vacuos chewing movements and inflammatory changes in mice via PPARγ receptors. Brain, Behavior, and Immunity, v. 74, p. 241-251, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.bbi.2018.09.014. Acesso em: 17 maio 2024.
APA
Sonego, A. B., Prado, D. S., Vale, G. T., Sepulveda-Diaz, J. E., Cunha, T. M., Tirapelli, C. R., et al. (2018). Cannabidiol prevents haloperidol-induced vacuos chewing movements and inflammatory changes in mice via PPARγ receptors. Brain, Behavior, and Immunity, 74, 241-251. doi:10.1016/j.bbi.2018.09.014
NLM
Sonego AB, Prado DS, Vale GT, Sepulveda-Diaz JE, Cunha TM, Tirapelli CR, Del Bel EA, Raisman-Vozari R, Guimarães FS. Cannabidiol prevents haloperidol-induced vacuos chewing movements and inflammatory changes in mice via PPARγ receptors [Internet]. Brain, Behavior, and Immunity. 2018 ; 74 241-251.[citado 2024 maio 17 ] Available from: https://doi.org/10.1016/j.bbi.2018.09.014
Vancouver
Sonego AB, Prado DS, Vale GT, Sepulveda-Diaz JE, Cunha TM, Tirapelli CR, Del Bel EA, Raisman-Vozari R, Guimarães FS. Cannabidiol prevents haloperidol-induced vacuos chewing movements and inflammatory changes in mice via PPARγ receptors [Internet]. Brain, Behavior, and Immunity. 2018 ; 74 241-251.[citado 2024 maio 17 ] Available from: https://doi.org/10.1016/j.bbi.2018.09.014
Artigo de periodico
Impairment of osmotic challenge-induced neurohypophyseal hormones secretion in sepsis survivor rats (2017)
Santos-Junior, Nilton Nascimento; Costa, Luís Henrique Angenendt; Catalão, Carlos Henrique Rocha; Kanashiro, Alexandre; Sharshar, Tarek; Rocha, Maria José Alves da
Fonte: Pituitary. Unidade: FORP
Assuntos: CITOCINAS, SEPSE, SISTEMA NERVOSO CENTRAL, VASOPRESSINAS, OCITOCINA
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SANTOS-JUNIOR, Nilton Nascimento et al. Impairment of osmotic challenge-induced neurohypophyseal hormones secretion in sepsis survivor rats. Pituitary, v. 20, n. 5, p. 515-521, 2017Tradução . . Disponível em: https://doi.org/10.1007/s11102-017-0812-z. Acesso em: 17 maio 2024.
APA
Santos-Junior, N. N., Costa, L. H. A., Catalão, C. H. R., Kanashiro, A., Sharshar, T., & Rocha, M. J. A. da. (2017). Impairment of osmotic challenge-induced neurohypophyseal hormones secretion in sepsis survivor rats. Pituitary, 20( 5), 515-521. doi:10.1007/s11102-017-0812-z
NLM
Santos-Junior NN, Costa LHA, Catalão CHR, Kanashiro A, Sharshar T, Rocha MJA da. Impairment of osmotic challenge-induced neurohypophyseal hormones secretion in sepsis survivor rats [Internet]. Pituitary. 2017 ; 20( 5): 515-521.[citado 2024 maio 17 ] Available from: https://doi.org/10.1007/s11102-017-0812-z
Vancouver
Santos-Junior NN, Costa LHA, Catalão CHR, Kanashiro A, Sharshar T, Rocha MJA da. Impairment of osmotic challenge-induced neurohypophyseal hormones secretion in sepsis survivor rats [Internet]. Pituitary. 2017 ; 20( 5): 515-521.[citado 2024 maio 17 ] Available from: https://doi.org/10.1007/s11102-017-0812-z
Artigo de periodico
Repurposing doxycycline for synucleinopathies: remodelling of α-synuclein oligomers towards non-toxic parallel beta-sheet structured species (2017)
González-Lizárraga, Florencia; Socías, Sergio B; Ávila, César L; Torres-Bugeau, Clarisa M; Barbosa, Leandro Ramos Souza; Binolfi, Andres; Sepúlveda-Díaz, Julia E; Del-Bel, Elaine; Fernandez, Claudio O; Papy-Garcia, Dulce; Itri, Rosangela; Raisman-Vozari Rita; Chehín, Rosana N
Fonte: Scientific Reports. Unidades: IF, FORP
Assuntos: DOENÇAS NEURODEGENERATIVAS, PROTEÍNAS, BIOFÍSICA
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ABNT
GONZÁLEZ-LIZÁRRAGA, Florencia et al. Repurposing doxycycline for synucleinopathies: remodelling of α-synuclein oligomers towards non-toxic parallel beta-sheet structured species. Scientific Reports, v. 7, n. 41755, p. 1-13, 2017Tradução . . Disponível em: https://doi.org/10.1038/srep41755. Acesso em: 17 maio 2024.
APA
González-Lizárraga, F., Socías, S. B., Ávila, C. L., Torres-Bugeau, C. M., Barbosa, L. R. S., Binolfi, A., et al. (2017). Repurposing doxycycline for synucleinopathies: remodelling of α-synuclein oligomers towards non-toxic parallel beta-sheet structured species. Scientific Reports, 7( 41755), 1-13. doi:10.1038/srep41755
NLM
González-Lizárraga F, Socías SB, Ávila CL, Torres-Bugeau CM, Barbosa LRS, Binolfi A, Sepúlveda-Díaz JE, Del-Bel E, Fernandez CO, Papy-Garcia D, Itri R, Raisman-Vozari Rita, Chehín RN. Repurposing doxycycline for synucleinopathies: remodelling of α-synuclein oligomers towards non-toxic parallel beta-sheet structured species [Internet]. Scientific Reports. 2017 ; 7( 41755): 1-13.[citado 2024 maio 17 ] Available from: https://doi.org/10.1038/srep41755
Vancouver
González-Lizárraga F, Socías SB, Ávila CL, Torres-Bugeau CM, Barbosa LRS, Binolfi A, Sepúlveda-Díaz JE, Del-Bel E, Fernandez CO, Papy-Garcia D, Itri R, Raisman-Vozari Rita, Chehín RN. Repurposing doxycycline for synucleinopathies: remodelling of α-synuclein oligomers towards non-toxic parallel beta-sheet structured species [Internet]. Scientific Reports. 2017 ; 7( 41755): 1-13.[citado 2024 maio 17 ] Available from: https://doi.org/10.1038/srep41755
Artigo de periodico
Novel tactics for neuroprotection in Parkinson's disease: role of antibiotics, polyphenols and neuropeptides (2016)
Reglodi, Dora; Renaud, Justine; Tamas, Andrea; Tizabi, Yousef; Socías, Sergio B; Del Bel, Elaine Aparecida; Raisman-Vozari , Rita
Fonte: Progress in Neurobiology. Unidade: FORP
Assuntos: DOENÇA DE PARKINSON, ANTIBIÓTICOS, ESTRESSE OXIDATIVO, ANTIOXIDANTES, TERAPÊUTICA, COMPOSTOS FENÓLICOS
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ABNT
REGLODI, Dora et al. Novel tactics for neuroprotection in Parkinson's disease: role of antibiotics, polyphenols and neuropeptides. Progress in Neurobiology, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.pneurobio.2015.10.004. Acesso em: 17 maio 2024.
APA
Reglodi, D., Renaud, J., Tamas, A., Tizabi, Y., Socías, S. B., Del Bel, E. A., & Raisman-Vozari , R. (2016). Novel tactics for neuroprotection in Parkinson's disease: role of antibiotics, polyphenols and neuropeptides. Progress in Neurobiology. doi:10.1016/j.pneurobio.2015.10.004
NLM
Reglodi D, Renaud J, Tamas A, Tizabi Y, Socías SB, Del Bel EA, Raisman-Vozari R. Novel tactics for neuroprotection in Parkinson's disease: role of antibiotics, polyphenols and neuropeptides [Internet]. Progress in Neurobiology. 2016 ;[citado 2024 maio 17 ] Available from: https://doi.org/10.1016/j.pneurobio.2015.10.004
Vancouver
Reglodi D, Renaud J, Tamas A, Tizabi Y, Socías SB, Del Bel EA, Raisman-Vozari R. Novel tactics for neuroprotection in Parkinson's disease: role of antibiotics, polyphenols and neuropeptides [Internet]. Progress in Neurobiology. 2016 ;[citado 2024 maio 17 ] Available from: https://doi.org/10.1016/j.pneurobio.2015.10.004
Artigo de periodico
CX3CR1 disruption differentially influences dopaminergic neuron degeneration in parkinsonian mice depending on the neurotoxin and route of administration (2016)
Tristão, Fabrine Sales Massafera; Lazzarini, Márcio; Martin, Sabine; Amar, Majid; Stühmer, Walter; Kirchhoff, Frank; Gomes, Lucas Araújo Caldi; Lanfumey, Laurance; Prediger, Rui D.; Sepulveda, Julia E.; Del Bel, Elaine Aparecida; Raisman-Vozari, Rita
Fonte: Neurotoxicity Research. Unidade: FORP
Assuntos: DOENÇA DE PARKINSON, INFLAMAÇÃO, DEGENERAÇÃO NEURAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
TRISTÃO, Fabrine Sales Massafera et al. CX3CR1 disruption differentially influences dopaminergic neuron degeneration in parkinsonian mice depending on the neurotoxin and route of administration. Neurotoxicity Research, v. 29, n. 3, p. 364-380, 2016Tradução . . Disponível em: https://doi.org/10.1007/s12640-015-9557-5. Acesso em: 17 maio 2024.
APA
Tristão, F. S. M., Lazzarini, M., Martin, S., Amar, M., Stühmer, W., Kirchhoff, F., et al. (2016). CX3CR1 disruption differentially influences dopaminergic neuron degeneration in parkinsonian mice depending on the neurotoxin and route of administration. Neurotoxicity Research, 29( 3), 364-380. doi:10.1007/s12640-015-9557-5
NLM
Tristão FSM, Lazzarini M, Martin S, Amar M, Stühmer W, Kirchhoff F, Gomes LAC, Lanfumey L, Prediger RD, Sepulveda JE, Del Bel EA, Raisman-Vozari R. CX3CR1 disruption differentially influences dopaminergic neuron degeneration in parkinsonian mice depending on the neurotoxin and route of administration [Internet]. Neurotoxicity Research. 2016 ; 29( 3): 364-380.[citado 2024 maio 17 ] Available from: https://doi.org/10.1007/s12640-015-9557-5
Vancouver
Tristão FSM, Lazzarini M, Martin S, Amar M, Stühmer W, Kirchhoff F, Gomes LAC, Lanfumey L, Prediger RD, Sepulveda JE, Del Bel EA, Raisman-Vozari R. CX3CR1 disruption differentially influences dopaminergic neuron degeneration in parkinsonian mice depending on the neurotoxin and route of administration [Internet]. Neurotoxicity Research. 2016 ; 29( 3): 364-380.[citado 2024 maio 17 ] Available from: https://doi.org/10.1007/s12640-015-9557-5
Artigo de periodico
Doxycycline suppresses microglial activation by inhibiting the p38 MAPK and NF-kB signaling pathways (2016)
Santa-Cecília, Flávia V.; Socias, Benjamin; Ouidja, Mohand O.; Sepulveda-Diaz, Julia E.; Acuña, Leonardo; Silva, Rangel L.; Michel, Patrick P.; Del Bel, Elaine Aparecida; Cunha, Thiago Mattar; Raisman-Vozari, Rita
Fonte: Neurotoxicity Research. Unidades: FORP, FMRP
Assuntos: DOENÇAS NEURODEGENERATIVAS, CITOCINAS, DOENÇA DE PARKINSON, FÁRMACOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANTA-CECÍLIA, Flávia V. et al. Doxycycline suppresses microglial activation by inhibiting the p38 MAPK and NF-kB signaling pathways. Neurotoxicity Research, v. 29, n. 4, p. 447-459, 2016Tradução . . Disponível em: https://doi.org/10.1007/s12640-015-9592-2. Acesso em: 17 maio 2024.
APA
Santa-Cecília, F. V., Socias, B., Ouidja, M. O., Sepulveda-Diaz, J. E., Acuña, L., Silva, R. L., et al. (2016). Doxycycline suppresses microglial activation by inhibiting the p38 MAPK and NF-kB signaling pathways. Neurotoxicity Research, 29( 4), 447-459. doi:10.1007/s12640-015-9592-2
NLM
Santa-Cecília FV, Socias B, Ouidja MO, Sepulveda-Diaz JE, Acuña L, Silva RL, Michel PP, Del Bel EA, Cunha TM, Raisman-Vozari R. Doxycycline suppresses microglial activation by inhibiting the p38 MAPK and NF-kB signaling pathways [Internet]. Neurotoxicity Research. 2016 ; 29( 4): 447-459.[citado 2024 maio 17 ] Available from: https://doi.org/10.1007/s12640-015-9592-2
Vancouver
Santa-Cecília FV, Socias B, Ouidja MO, Sepulveda-Diaz JE, Acuña L, Silva RL, Michel PP, Del Bel EA, Cunha TM, Raisman-Vozari R. Doxycycline suppresses microglial activation by inhibiting the p38 MAPK and NF-kB signaling pathways [Internet]. Neurotoxicity Research. 2016 ; 29( 4): 447-459.[citado 2024 maio 17 ] Available from: https://doi.org/10.1007/s12640-015-9592-2
Artigo de periodico
Evaluation of nigrostriatal neurodegeneration and neuroinflammation following repeated intranasal 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) administration in mice, an experimental model of Parkinson's disease (2014)
Tristão, Fabrine S. M.; Amar, Majid; Latrous, Ines; Del Bel, Elaine Aparecida; Prediger, Rui D.; Raisman-Vozari, Rita
Fonte: Neurotoxicity Research. Unidade: FORP
Assuntos: MODELOS ANIMAIS DE DOENÇAS, EXPOSIÇÃO AMBIENTAL, DOENÇAS NEURODEGENERATIVAS (PATOLOGIA)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
TRISTÃO, Fabrine S. M. et al. Evaluation of nigrostriatal neurodegeneration and neuroinflammation following repeated intranasal 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) administration in mice, an experimental model of Parkinson's disease. Neurotoxicity Research, v. 25, n. 1, p. 24-32, 2014Tradução . . Disponível em: https://doi.org/10.1007/s12640-013-9401-8. Acesso em: 17 maio 2024.
APA
Tristão, F. S. M., Amar, M., Latrous, I., Del Bel, E. A., Prediger, R. D., & Raisman-Vozari, R. (2014). Evaluation of nigrostriatal neurodegeneration and neuroinflammation following repeated intranasal 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) administration in mice, an experimental model of Parkinson's disease. Neurotoxicity Research, 25( 1), 24-32. doi:10.1007/s12640-013-9401-8
NLM
Tristão FSM, Amar M, Latrous I, Del Bel EA, Prediger RD, Raisman-Vozari R. Evaluation of nigrostriatal neurodegeneration and neuroinflammation following repeated intranasal 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) administration in mice, an experimental model of Parkinson's disease [Internet]. Neurotoxicity Research. 2014 ; 25( 1): 24-32.[citado 2024 maio 17 ] Available from: https://doi.org/10.1007/s12640-013-9401-8
Vancouver
Tristão FSM, Amar M, Latrous I, Del Bel EA, Prediger RD, Raisman-Vozari R. Evaluation of nigrostriatal neurodegeneration and neuroinflammation following repeated intranasal 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) administration in mice, an experimental model of Parkinson's disease [Internet]. Neurotoxicity Research. 2014 ; 25( 1): 24-32.[citado 2024 maio 17 ] Available from: https://doi.org/10.1007/s12640-013-9401-8
Artigo de periodico
Differential transcript profiles of MHC class Ib (Qa-1, Qa-2, and Qa-10) and Aire genes during the ontogeny of thymus and other tissues (2014)
Melo-Lima, Breno Luiz; Evangelista, Adriane Feijó; Magalhães, Danielle Aparecida Rosa de; Passos Junior, Geraldo Aleixo da Silva; Moreau, Philipe; Donadi, Eduardo Antônio
Fonte: Journal of Immunology Research. Unidades: FORP, FMRP
Assuntos: ONTOGENIA, TRANSCRIÇÃO GÊNICA, EXPRESSÃO GÊNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MELO-LIMA, Breno Luiz et al. Differential transcript profiles of MHC class Ib (Qa-1, Qa-2, and Qa-10) and Aire genes during the ontogeny of thymus and other tissues. Journal of Immunology Research, v. 2014, 2014Tradução . . Disponível em: https://doi.org/10.1155/2014/159247. Acesso em: 17 maio 2024.
APA
Melo-Lima, B. L., Evangelista, A. F., Magalhães, D. A. R. de, Passos Junior, G. A. da S., Moreau, P., & Donadi, E. A. (2014). Differential transcript profiles of MHC class Ib (Qa-1, Qa-2, and Qa-10) and Aire genes during the ontogeny of thymus and other tissues. Journal of Immunology Research, 2014. doi:10.1155/2014/159247
NLM
Melo-Lima BL, Evangelista AF, Magalhães DAR de, Passos Junior GA da S, Moreau P, Donadi EA. Differential transcript profiles of MHC class Ib (Qa-1, Qa-2, and Qa-10) and Aire genes during the ontogeny of thymus and other tissues [Internet]. Journal of Immunology Research. 2014 ; 2014[citado 2024 maio 17 ] Available from: https://doi.org/10.1155/2014/159247
Vancouver
Melo-Lima BL, Evangelista AF, Magalhães DAR de, Passos Junior GA da S, Moreau P, Donadi EA. Differential transcript profiles of MHC class Ib (Qa-1, Qa-2, and Qa-10) and Aire genes during the ontogeny of thymus and other tissues [Internet]. Journal of Immunology Research. 2014 ; 2014[citado 2024 maio 17 ] Available from: https://doi.org/10.1155/2014/159247
Artigo de periodico
Integrative analysis of the transcriptome profiles observed in type 1, type 2 and gestational diabetes mellitus reveals the role of inflammation (2014)
Evangelista, Adriane F.; Collares, Christianna V. A.; Xavier, Danilo J.; Macedo, Claudia; Manoel-Caetano, Fernanda S.; Rassi, Diane M.; Foss-Freitas, Maria Cristina; Foss, Milton César; Sakamoto-Hojo, Elza Tiemi; Nguyen, Catherine; Puthier, Denis; Passos, Geraldo Aleixo Silva; Donadi, Eduardo Antônio
Fonte: BMC Medical Genomics. Unidades: FMRP, FFCLRP, FORP
Assuntos: DIABETES MELLITUS, GRAVIDEZ NO DIABETES, IMUNOLOGIA, INFLAMAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
EVANGELISTA, Adriane F. et al. Integrative analysis of the transcriptome profiles observed in type 1, type 2 and gestational diabetes mellitus reveals the role of inflammation. BMC Medical Genomics, v. 7, 2014Tradução . . Disponível em: https://doi.org/10.1186/1755-8794-7-28. Acesso em: 17 maio 2024.
APA
Evangelista, A. F., Collares, C. V. A., Xavier, D. J., Macedo, C., Manoel-Caetano, F. S., Rassi, D. M., et al. (2014). Integrative analysis of the transcriptome profiles observed in type 1, type 2 and gestational diabetes mellitus reveals the role of inflammation. BMC Medical Genomics, 7. doi:10.1186/1755-8794-7-28
NLM
Evangelista AF, Collares CVA, Xavier DJ, Macedo C, Manoel-Caetano FS, Rassi DM, Foss-Freitas MC, Foss MC, Sakamoto-Hojo ET, Nguyen C, Puthier D, Passos GAS, Donadi EA. Integrative analysis of the transcriptome profiles observed in type 1, type 2 and gestational diabetes mellitus reveals the role of inflammation [Internet]. BMC Medical Genomics. 2014 ; 7[citado 2024 maio 17 ] Available from: https://doi.org/10.1186/1755-8794-7-28
Vancouver
Evangelista AF, Collares CVA, Xavier DJ, Macedo C, Manoel-Caetano FS, Rassi DM, Foss-Freitas MC, Foss MC, Sakamoto-Hojo ET, Nguyen C, Puthier D, Passos GAS, Donadi EA. Integrative analysis of the transcriptome profiles observed in type 1, type 2 and gestational diabetes mellitus reveals the role of inflammation [Internet]. BMC Medical Genomics. 2014 ; 7[citado 2024 maio 17 ] Available from: https://doi.org/10.1186/1755-8794-7-28
Artigo de periodico
GM-CSF priming drives bone marrow-derived macrophages to a pro-inflammatory pattern and downmodulates PGE2 in response to TLR2 ligands (2012)
Sorgi, Carlos Arterio ; Rose, Stephanie; Court, Nathalie; Carlos, Daniela ; Paula-Silva, Francisco Wanderley Garcia de ; Assis, Patricia Aparecida de; Frantz, Fabiani Gai ; Ryffel, Bernhard; Quesniaux, Valerie; Faccioli, Lucia Helena
Fonte: PLOS ONE. Unidades: FCFRP, FMRP, FORP
Assuntos: MACRÓFAGOS, CITOCINAS, SISTEMA IMUNE, INFLAMAÇÃO, MEDULA ÓSSEA, LIGANTES, IMUNOLOGIA, RECEPTORES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SORGI, Carlos Arterio et al. GM-CSF priming drives bone marrow-derived macrophages to a pro-inflammatory pattern and downmodulates PGE2 in response to TLR2 ligands. PLOS ONE, v. 7, n. 7, p. 1-13, 2012Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0040523. Acesso em: 17 maio 2024.
APA
Sorgi, C. A., Rose, S., Court, N., Carlos, D., Paula-Silva, F. W. G. de, Assis, P. A. de, et al. (2012). GM-CSF priming drives bone marrow-derived macrophages to a pro-inflammatory pattern and downmodulates PGE2 in response to TLR2 ligands. PLOS ONE, 7( 7), 1-13. doi:10.1371/journal.pone.0040523
NLM
Sorgi CA, Rose S, Court N, Carlos D, Paula-Silva FWG de, Assis PA de, Frantz FG, Ryffel B, Quesniaux V, Faccioli LH. GM-CSF priming drives bone marrow-derived macrophages to a pro-inflammatory pattern and downmodulates PGE2 in response to TLR2 ligands [Internet]. PLOS ONE. 2012 ; 7( 7): 1-13.[citado 2024 maio 17 ] Available from: https://doi.org/10.1371/journal.pone.0040523
Vancouver
Sorgi CA, Rose S, Court N, Carlos D, Paula-Silva FWG de, Assis PA de, Frantz FG, Ryffel B, Quesniaux V, Faccioli LH. GM-CSF priming drives bone marrow-derived macrophages to a pro-inflammatory pattern and downmodulates PGE2 in response to TLR2 ligands [Internet]. PLOS ONE. 2012 ; 7( 7): 1-13.[citado 2024 maio 17 ] Available from: https://doi.org/10.1371/journal.pone.0040523

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